Complement activity is regulated in C3 glomerulopathy by IgG–factor H fusion proteins with and without properdin targeting domains - ScienceDirect

PDF) Complement-mediated kidney diseases

Complement activity is regulated in C3 glomerulopathy by IgG–factor H fusion proteins with and without properdin targeting domains - ScienceDirect

PDF) Developments in anti-complement therapy; from disease to clinical trial

Complement in metabolic disease: metaflammation and a two-edged sword

Anti-complement-factor H-associated glomerulopathies

MAGI-2 orchestrates the localization of backbone proteins in the slit diaphragm of podocytes - Kidney International

C3 glomerulopathies (C3G) are caused by dysregulation of the

C3 glomerulopathy and atypical hemolytic uremic syndrome: an updated review of the literature on alternative complement pathway disorders

Complement-mediated kidney diseases - ScienceDirect

Recurrent C3 glomerulopathy after kidney transplantation - ScienceDirect